Tablysin-15 inhibits osteoclastogenesis and LPS-induced bone loss via attenuating the integrin αvβ3 pathway - ScienceDirect
Loss of Protein Kinase C-δ Protects against LPS-Induced Osteolysis Owing to an Intrinsic Defect in Osteoclastic Bone Resorption | PLOS ONE
Frontiers | Macrophage-Osteoclast Associations: Origin, Polarization, and Subgroups
Lipopolysaccharide- TLR-4 Axis regulates Osteoclastogenesis independent of RANKL/RANK signaling | BMC Immunology | Full Text
Schematic illustration of the consecutive phases of osteoclast... | Download Scientific Diagram
LPS induces osteoclast differentiation in RANKL-primed cells. a The... | Download Scientific Diagram
Artemether attenuates LPS-induced inflammatory bone loss by inhibiting osteoclastogenesis and bone resorption via suppression of MAPK signaling pathway | Cell Death & Disease
DHA inhibits LPS-induced osteoclast differentiation. (a) Chemical... | Download Scientific Diagram
The effect of lipoxin A4 on E. coli LPS-induced osteoclastogenesis | SpringerLink
IJMS | Free Full-Text | NLRP3 Inflammasome Negatively Regulates RANKL-Induced Osteoclastogenesis of Mouse Bone Marrow Macrophages but Positively Regulates It in the Presence of Lipopolysaccharides
Pam 2 and Pam3 stimulate mature osteoclast formation, whereas S. aureus, LPS P. gingivalis and HKLM stimulate differentiation of mononuclear osteoclasts in RANKL-primed bone marrow macrophage cultures.
Umbelliferone Prevents Lipopolysaccharide-Induced Bone Loss and Suppresses RANKL-Induced Osteoclastogenesis by Attenuating Akt-c-Fos-NFATc1 Signaling
Betulinic Acid Inhibits RANKL-Induced Osteoclastogenesis via Attenuating Akt, NF-κB, and PLCγ2-Ca2+ Signaling and Prevents Inflammatory Bone Loss | Journal of Natural Products
Marine Drugs | Free Full-Text | Fucoidan Prevents RANKL-Stimulated Osteoclastogenesis and LPS-Induced Inflammatory Bone Loss via Regulation of Akt/GSK3β/PTEN/NFATc1 Signaling Pathway and Calcineurin Activity
Analysis of RANK expression in LPS- and RANKL-mediated... | Download Scientific Diagram
Hesperetin suppresses RANKL‐induced osteoclastogenesis and ameliorates lipopolysaccharide‐induced bone loss - Liu - 2019 - Journal of Cellular Physiology - Wiley Online Library
Figure 1 from Lipopolysaccharide (LPS) promotes osteoclast differentiation and activation by enhancing the MAPK pathway and COX-2 expression in RAW264.7 cells. | Semantic Scholar
Lipopolysaccharide- TLR-4 Axis regulates Osteoclastogenesis independent of RANKL/RANK signaling | BMC Immunology | Full Text
RO4929097 regulates RANKL-induced osteoclast formation and LPS-mediated bone resorption | Aging
Osteoclastogenesis in periodontal diseases: Possible mediators and mechanisms. - Abstract - Europe PMC
Role of Muramyl Dipeptide in Lipopolysaccharide-Mediated Biological Activity and Osteoclast Activity
Artesunate attenuates LPS-induced osteoclastogenesis by suppressing TLR4/TRAF6 and PLCγ1-Ca2+-NFATc1 signaling pathway | Acta Pharmacologica Sinica
IJMS | Free Full-Text | Osteoclast Multinucleation: Review of Current Literature
IJMS | Free Full-Text | Synovial Fluid Interleukin-16 Contributes to Osteoclast Activation and Bone Loss through the JNK/NFATc1 Signaling Cascade in Patients with Periprosthetic Joint Infection
